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Telomeres function similarly, preventing chromosomes from fraying or tangling with one another. When that happens, it can cause genetic information to get mixed up or destroyed, leading to cell malfunction, increasing the risk of disease or even shortening lifespan.\xa0 Each time a cell divides, its telomeres become shorter. After years of splicing and dicing, telomeres become too short for more divisions. At this point, cells are unable to divide further and become inactive, or die.\n\nAs more of our cells lose their telomeres and go out of commission, without others to take their place, the body follows and begins breaking down.\xa0 Telomeres don\u2019t leave (or shorten) quietly. Their shortening process has been linked with aging, cancer and a higher risk of death. A study found that individuals with the shortest telomeres, or about 10 percent of the study\u2019s participants, were 23 percent more likely to die within three years than those with longer telomeres.\xa0 There is an enzyme called\xa0Telomerase that lengthens telomeres and keeps them from wearing out too fast or too early, which helps you live longer. \xa0By reawakening telomerase in human cells where it\u2019s stopped working, normal human aging could be slowed. Want to start Aging Younger? Tune in Wednesday JUNE 12TH at 7PM FOR THE REST OF THE STORY!\n\nhttp://tobtr.com/s/11375979